Chapter 48. Multifetal Pregnancy. Will Obs.

 Multifetal Pregnancy

BS. Nguyễn Hồng Anh

Multietal pregnancies may result rom two or more ertilization events, rom a single ertilization ollowed by a splitting o the zygote, or both. In part because o inertility therapy, the twin birth rate in the United States was 3.2 percent in 2019. For higher-order multietal births, the number peaked in 1998

at 0.2 percent o all births. Subsequent eorts in the reproductive endocrinology community to curb this rate have led to

declines. Specically, the rate o triplets or more declined by 55

percent rom 1998 to 2019 (Martin, 2021).

Multietal gestations remain problematic or both the mother

and her etuses. For the etus, multietal births accounted or

3 percent o all live births in 2013 but or 15 percent o all

inant deaths. Specically, the inant mortality rate or twins

was more than our times the rate or singletons, and or triplets

it was 12-old higher (Matthews, 2015). Neonates rom multi-

etal gestations also make up a disproportionate percentage o

very-low-birthweight newborns in the United States (Martin,

2021). Tese two outcomes in singletons and twins rom Parkland Hospital are shown in Table 48-1. Risks or congenital

malormations and preterm birth are greater with multietal

gestation and discussed later.

Te mother may also experience higher morbidity and mortality rates, and these rise with the number o etuses (Society

or Maternal-Fetal Medicine, 2019). In one study o more than

44,000 multietal pregnancies, the risks or preeclampsia, postpartum hemorrhage, and maternal death were twoold higher

than these rates in singleton gestations (Walker, 2004). Rates

o placenta previa and placenta accreta spectrum are increased

(Miller, 2021a). Moreover, compared with rates or singletons

the peripartum hysterectomy rate was threeold greater or

twins and 24-old higher or triplets or quadruplets in one

study (Francois, 2005).

MECHANISMS OF MULTIFETAL GESTATIONS

■ Dizygotic versus Monozygotic Twinning

Multietal gestations are oten described by zygosity, amnionicity, and chorionicity, which are the number o zygotes, amnions,

and chorions, respectively. win etuses most oten result rom

ertilization o two separate ova, which yields dizygotic or raternal twins. Less oten, twins arise rom a single ertilized ovum

that then divides to create monozygotic or identical twins.

Dizygotic twins are not in a strict sense true twins because

they result rom the maturation and ertilization o two ova

during a single ovulatory cycle. Genetically, these twins are like

any other pair o siblings. wins o opposite sex are almost

always dizygotic. Rarely, somatic mutations or chromosome

aberration in one monozygotic twin can yield diering gender

karyotype or phenotype. For example, postzygotic loss o the Y

chromosome in one 46,XY twin creates a phenotypically emale

twin with 45,X urner syndrome.

Monozygotic twins, although they have the same genetic

heritage, also are usually not identical. For example, the division o one ertilized zygote into two does not necessarily yield

equal sharing o protoplasmic material. In addition, monozygotic twins may be discordant or genetic mutations because

o a postzygotic mutation, or they may have the same genetic

disease but with marked variability in its expression. In emale

etuses, skewed lyonization can produce dierential expression o X-linked traits or diseases. Last, sesquizygosity is a rare

event o dispermic ertilization that leads to monozygotic twins.

Some cases may have sex discordance (Gabbett, 2019).

■ Monozygotic Twinning

Mechanisms underlying monozygotic twinning are poorly

understood. One association is assisted reproductive technology (AR) and in-vitro ertilization (IVF). Te monozygotic

twinning incidence is twoold greater in pregnancies conceived

using blastocyst transer compared with transer o a later-stage

embryo. Te predisposition to splitting may stem rom culture

media eects (Busnelli, 2019).

Te outcome o the monozygotic twinning process depends

on when division occurs as depicted in Figure 48-1. I zygotes

divide within the rst 72 hours ater ertilization, two embryos,

two amnions, and two chorions develop, and a monozygotic,

dichorionic, diamnionic twin gestation evolves. wo distinct

placentas or a single, used placenta may develop. I division

occurs between days 4 through 8, a monozygotic, monochorionic, diamnionic twin pregnancy results. By 8 days, the chorion and the amnion have already dierentiated, and division

results in two embryos within a common amnionic sac, which

is a monozygotic, monochorionic, monoamnionic twin gestation.

Conjoined twins result i division initiates later.

Rarely, monochorionic twins may be dizygotic. Mechanisms

are unclear. In one review o 31 cases, nearly 80 percent were

conceived ater AR procedures, and 90 percent were associated with chimerism (Peters, 2017).

■ Factors Affecting Twinning

Dizygotic twinning is much more common than monozygotic

splitting o a single oocyte, and its incidence is positively inuenced by inertility treatment and by maternal age, race, heredity, and size. By contrast, the requency o monozygotic twin

births is relatively constant worldwide—approximately one set

per 250 births. Tis incidence is generally independent o most

demographic actors, except AR.

O inertility therapies, ovulation induction with clomiphene

citrate or with ollicle-stimulating hormone (FSH) plus human

chorionic gonadotropin (hCG) remarkably enhances the likelihood o multiple concurrent ovulations. Moreover, with IVF,

the greater the number o embryos that are transerred, the

greater the risk o twins or more. In 2017, AR contributed to

1.9 percent o all newborns in the United States and to 14.7 percent o all neonates in multietal gestations (Sunderam, 2020).

Te American Society or Reproductive Medicine (2017)

revised their guidelines regarding the number o embryos or

blastocysts transerred during IVF to reduce the incidence o

multietal pregnancies. For example, women younger than 35

years are encouraged to receive a single-embryo transer, regardless o embryo stage. Tese practices have eectively lowered

multietal rates (Martin, 2021).

Advancing maternal age and delayed childbearing are other

important risk actors (Adashi, 2018; Otta, 2016). One explanation is FSH levels, which rise with age and lead to greater

ovarian stimulation (Beemsterboer, 2006). Higher levels o

FSH have been linked with dizygotic twinning (Lambalk,

1998). Another explanation may be a higher use o AR in

older women. Paternal age has also been linked to twinning,

but its eect is elt to be small (Abel, 2012).

Diferent races and ethnic groups vary in their requency o

multietal births. In the United States in 2019, the twin birth

rate was 4.1 percent in black women, 3.3 percent in whites,

and 2.5 in Hispanics (Martin, 2021). In one rural community

in Nigeria, twinning occurred once in every 20 births (Knox,

1960). Tese marked dierences in twinning requency may

stem rom racial variations in FSH levels (Nylander, 1973).

Heredity is another actor, and maternal inuence appears

to supersede that o the ather. In a study o 4000 genealogical records, women who themselves were a dizygotic twin gave

birth to twins at a rate o one set per 58 births (White, 1964).

Women who were not a twin, but whose husbands were a dizygotic twin, gave birth to twins at a rate o one set per 116

pregnancies. Genome-wide association studies have identied

potentially contributory genes, and two are related to FSH

(Mbarek, 2016). However, the contribution o these variants

to the overall incidence o twinning is likely small (Hoekstra,

2008).

Maternal size is another risk actor. Nylander (1971) showed

an increasing gradient in the twinning rate in taller, heavier

women. Tese had a twinning rate 25 to 30 percent higher

than short, nutritionally deprived women. Likewise, another

study ound an association between greater maternal weight

and dizygotic twinning (Reddy, 2005). Evidence acquired during and ater World War II suggests that twinning correlates

more with nutrition than with body size. Widespread undernourishment in Europe during those years was associated with

a marked all in the dizygotic twinning rate (Bulmer, 1959).

■ Superfecundation and Superfetation

Superecundation is ertilization o two ova within the same

menstrual cycle but not at the same coitus nor necessarily by

sperm rom the same male. Te latter leads to heteropaternity

(Silver, 2021).

In superetation, an interval as long as or longer than a menstrual cycle intervenes between ertilizations. Superetation is

not known to occur spontaneously and is likely due to AR

(Lantieri, 2010). Pseudo-superetation oten results rom markedly unequal growth o twins with the same gestational age.

DIAGNOSIS OF MULTIFETAL GESTATION

■ Clinical Evaluation

Early diagnosis can help with management o the associated

risks posed by twins. Accurate undal height measurement can

be an initial tool. With multiples, uterine size is typically larger

during the second trimester than that expected or a singleton. In one study, undal heights obtained between 20 and 30

weeks’ gestation averaged 5 cm greater in twins than in singletons (Rouse, 1993).

Palpation o etal parts to diagnose twins beore the third

trimester is difcult. Even then, obesity or hydramnios can hinder assessment. Palpating two etal heads strongly supports the

diagnosis. Moreover, a hand-held Doppler ultrasonic unit may

isolate two etal heartbeats i their rates are clearly distinct rom

each other and rom the mother.

Overall, however, clinical criteria alone to diagnose multietal

gestations is unreliable. In the Routine Antenatal Diagnostic Imaging with Ultrasound (RADIUS) trial, or 37 percent o women

who did not have a screening ultrasound examination, their twin

pregnancy was not diagnosed until 26 weeks’ gestation. In 13 percent o unscanned women, their multietal gestation was identied

only during their admission or delivery (LeFevre, 1993).

■ Sonography

Sonographic examination should detect practically all sets o

twins. Further, it should aim to determine etal number, estimated

gestational age, chorionicity, and amnionicity. Importantly, the

risk or many twin-specic complications varies in relation to

these (Table 48-2) (Hack, 2008; Manning, 1995).

With sonography, separate gestational sacs, i present, can

be identied early in twin pregnancy (Fig. 48-2). Subsequently,

each etal head should be seen in two perpendicular planes to

avoid mistaking a etal trunk or a second etal head. Ideally,

two etal heads or two abdomens are seen in the same image

plane to avoid scanning the same etus twice and interpreting

it as twins. Higher-order multietal gestations are more challenging to evaluate. Even in the rst trimester, identiying the

actual number o etuses and their position can be difcult.

Tis determination is especially important i selective reduction

is considered (p. 858).

In determining chorionicity, sonography’s accuracy diminishes as gestational age advances. It has a 98-percent accuracy

in the rst trimester but may be incorrect in up to 10 percent

o second-trimester examinations. Overall, chorionicity can

be correctly determined with sonography beore 24 weeks in

approximately 95 percent o cases (Emery, 2015; Lee, 2006).

Te sonographic eatures that are used to determine chorionicity vary according to gestational age. Early in the rst

trimester, the number o chorions equates to the number o

gestational sacs. A thick band o chorion that separates two

gestational sacs signals a dichorionic pregnancy, whereas monochorionic twins have a single gestational sac. I the gestation is

monochorionic diamnionic, it may be difcult to visualize the

thin intervening amnion beore 8 weeks’ gestation. I the intervening membrane is difcult to visualize, the number o yolk

sacs usually correlates with the number o amnions. However,

the number o yolk sacs as a predictor o amnionicity may not

always be accurate (Shen, 2006). Although uncommonly seen

early, cord entanglement identies a monoamnionic gestation.

At 10 to 14 weeks’ gestation, sonographic assessment o

chorionicity may be determined using our eatures. Tese are

the number o placental masses, presence o an intervening

membrane dividing the sacs, thickness o that membrane, and

etal gender (Emery, 2015; Khalil, 2016). First, two separate

placentas suggest dichorionicity. Te converse is not necessarily true, such as cases with a single used placental mass. Second, identication o a thick dividing membrane—generally

≥2 mm—supports a presumed diagnosis o dichorionicity. In

a dichorionic pregnancy, this visualized membrane is composed

o our layers—two amnions and two chorions. Also, the twin

peak sign—also called lambda or delta sign—is seen by examining the point o origin o the dividing membrane on the placental surace. Te peak appears as a triangular projection o

placental tissue extending a short distance between the layers o

the dividing membrane (Fig. 48-3).

In contrast, monochorionic pregnancies have a dividing

membrane that is so thin (generally <2 mm) that it may not

be seen until the second trimester. Te relationship between

the membranes and placenta without apparent extension o

placenta between the dividing membranes is called the T sign

(Fig. 48-4). Evaluation o the dividing membrane can establish

chorionicity in more than 99 percent o pregnancies in the rst

trimester (Maruotti, 2016; Miller, 2012). Lack o a dividing

membrane signals a monochorionic monoamnionic gestation.

Last, twins with diering gender indicates a dichorionic

(and dizygotic) gestation. Rare exceptions were described earlier (p. 839).

■ Other Diagnostic Aids

Magnetic resonance (MR) imaging may help delineate complications in monochorionic twins, including conjoined twins

(Hu, 2006). Abdominal radiography can be used i etal number in a higher-order multietal gestation is uncertain. However, radiographs generally have limited utility and lead to an

incorrect diagnosis i etuses move during the exposure.

No biochemical test reliably identies multietal gestations.

Serum levels o β-hCG and o maternal serum alpha-etoprotein (MSAFP) are usually higher, but ranges may overlap with

those o singletons.

■ Placental Examination

Careul visual examination o the placenta and membranes ater

delivery can establish zygosity and chorionicity in many cases.

First, the placenta is gently delivered to preserve the attachment o the amnion and chorion. With one common amnionic

sac or with juxtaposed amnions not separated by chorion, the

etuses are monozygotic (see Fig. 48-1). I adjacent amnions

are separated by chorion, the etuses could be either dizygotic

or monozygotic, but dizygosity is more common (Fig. 48-5).

I the neonates are o the same sex, blood typing o cord blood

samples may be helpul. Dierent genders or blood types

reects dizygosity, although the same gender or blood type in

each etus does not conrm monozygosity. Postnatal zygosity

genetic testing is available, and the benets and ethics have

been debated (Brown, 2015; Craig, 2015).

MATERNAL PHYSIOLOGICAL ADAPTATIONS

Te physiological burdens o pregnancy and likelihood o serious maternal complications are typically greater with multietal

gestations than with singleton ones. In the rst trimester and

with its higher serum β-hCG levels, multietal gestations oten

cause nausea and vomiting. In women carrying more than one

etus, blood volume expansion is greater and averages 50 to

60 percent compared with 40 to 50 percent in those with a

singleton (Pritchard, 1965). Tis augmented hypervolemia

teleologically osets blood loss with vaginal delivery o twins,

which is twice that with a single etus. Although red cell mass

also accrues, it does so proportionately less in twin pregnancies. Combined with greater iron and olate requirements, this

predisposes to anemia.

Women carrying twins also have a typical pattern o blood

pressure change. One study assessed serial blood pressures in

more than 13,000 singleton and twin pregnancies (MacDonaldWallis, 2012). As early as 8 weeks’ gestation, the diastolic blood

pressure in women with twins was lower than that with singleton pregnancies. It generally rose by a greater degree at term. An

earlier study demonstrated that this later rise was at least 15 mm

Hg in 95 percent o women with twins compared with only 54

percent o women with a singleton (Campbell, 1986).

Hypervolemia along with decreased vascular resistance has

an impressive eect on cardiac unction. In one study o 119

women with twins, cardiac output rose another 20 percent

above that in women with a singleton (Kametas, 2003). Similarly, a study o serial maternal echocardiography examinations

ound a greater elevation in cardiac output in 20 women with

uncomplicated twin pregnancies (Kuleva, 2011). Both studies

ound that the augmented cardiac output was predominantly

due to greater stroke volume rather than higher heart rate.

Vascular resistance was signicantly lower in twin gestations

throughout pregnancy compared with singleton ones. In a

study o 30 uncomplicated twin pregnancies, this same group

o investigators identied progressive diastolic dysunction

rom the rst to third trimester. Te dysunction subsequently

normalized ater delivery (Ghi, 2015).

Uterine growth in a multietal gestation is substantively

greater than in a singleton one. Te uterus and its nonetal

contents may achieve a volume o 10 L or more and weigh

in excess o 20 pounds. Especially with monozygotic twins,

excessive amounts o amnionic uid may rapidly accumulate.

In these circumstances, maternal abdominal viscera and lungs

can be appreciably compressed and displaced by the expanding

uterus. As a result, the size and weight o the large uterus may

preclude more than a sedentary existence or these women.

Rarely, maternal renal unction can become seriously

impaired, most likely as the consequence o obstructive uropathy (Jena, 1996). Hydramnios is a common associate, and therapeutic amniocentesis may provide relie or the mother, may

improve obstructive uropathy, and possibly may lower rates o

preterm labor or rupture o membranes. Unortunately, hydramnios oten develops remote rom term and rapidly reaccumulates.

PREGNANCY COMPLICATIONS

Several complications, discussed next, complicate multietal pregnancy. Others are preterm birth and discordant etal

growth, which are discussed in later sections.

■ Spontaneous Abortion and Vanishing Fetus

Among twins, monochorionic twins have signicantly higher

early etal loss rates than dichorionic pairs (D’Antonio, 2013)

wins achieved through AR may be at greater risk or abortion compared with those conceived spontaneously (Szymusik,

2012). However, among AR-conceived gestations, twins have

lower miscarriage rates than singletons (Matias, 2007).

In some cases, only one etus is spontaneously lost. As a

result, the incidence o twins in the rst trimester is much

greater than the incidence o twins at birth. Estimates suggest

that while 1 in 8 pregnancies begin multietal, only 1 in 80

births are multietal (Corsello, 2010). Sonography studies in

the rst trimester have shown that one twin dies and “vanishes”

beore the second trimester in 10 to 40 percent o all twin pregnancies (Brady, 2013; Harris, 2020). Te incidence is higher

ollowing AR conception.

A vanishing etus is more common in higher-order multiples. In one study o spontaneous reduction in 709 multietal

pregnancies beore 12 weeks’ gestation, one or more embryos

died in 36 percent o twin pregnancies, in 53 percent o triplet pregnancies, and in 65 percent o quadruplet pregnancies

(Dickey, 2002). Interestingly, in one study, ultimate pregnancy

duration and birthweight were inversely related to initial gestational sac number regardless o the nal number o etuses

at delivery (Seong, 2020). Tis eect was most pronounced in

twins who started as quadruplets. Evidence or adverse immediate and long-term eects o twin spontaneous reduction on the

remaining pregnancy is conicting (Harris, 2020; McNamara,

2016).

Spontaneous reduction o a twin gestation may aect prenatal screening results. In one study o AR-conceived gestations,

56 twin pregnancies with a single early demise and 897 singleton gestations were compared (Gjerris, 2009). First-trimester

serum marker concentrations did not dier between the groups

i the embryonic loss was identied beore 9 weeks’ gestation. I

diagnosed ater 9 weeks, the marker levels were higher and less

precise. With a vanishing twin, rst-trimester maternal serum

levels o the pregnancy-associated plasma protein A (PAPP-A)

can be elevated. Second-trimester MSAFP and dimeric inhibin

A levels also can be higher (Huang, 2015). Early loss o one

twin may also aect noninvasive prenatal testing using cell-ree

DNA (cDNA) (Chap. 17, p. 336). In one report, 15 percent

o the alse-positive results were attributed to this eect (Futch,

2013). Te development o single-nucleotide polymorphism

technology or cDNA testing holds promise in better identiying these cases (Curnow, 2015). Tus, a spontaneously reduced

abortus is ideally identied to help avoid conusion with results

rom aneuploidy and neural-tube deect screening.

■ Congenital Malformations

Te incidence o these is appreciably higher in multietal gestations compared with singleton ones. In one study, the congenital malormation rate was 406 per 10,000 twins compared

with 238 per 10,000 singletons (Glinianaia, 2008). Tis rate

in monochorionic twins was almost twice that o dichorionic

twin gestations. One large study between 1998 and 2010 ound

that twins had a 73-percent greater risk o congenital heart disease than singletons. Te occurrence risk and concordance were

substantially higher among monochorionic twins (Best, 2015;

Gijtenbeek, 2019).

From a 30-year European registry o multietal births, structural anomaly rates rose steadily rom 2.2 percent in 1987 to

3.3 percent in 2007 (Boyle, 2013). Yet, during this time, the

proportion o dizygotic twins grew by 30 percent, whereas the

proportion o monozygotic twins remained stable. Tis higher

risk o congenital malormations in dizygotic twins over time

correlated with increased availability o AR. An increase in

birth deect rates related to AR has been reported (Wen,

2020). However, i data are adjusted or maternal age or duration o subertility, the risk o congenital anomalies does not

appear to be increased by AR (Zhu, 2006).

■ Low Birthweight

Multietal gestations are more likely to be low birthweight

than singleton pregnancies due to restricted etal growth and

preterm delivery. Birthweights in twins closely paralleled those

o singletons until 28 to 30 weeks’ gestation. Tereater, twin

birthweights progressively lagged (Fig. 48-6). Beginning at 35

to 36 weeks’ gestation, twin birthweights clearly diverge rom

those o singletons. Tus, abnormal growth should be diagnosed only when etal size is less than expected or multietal

gestation. Accordingly, twin and triplet growth curves have been

developed (Kim, 2010; Odibo, 2013; Vora, 2006). o conrm

suitable growth in dichorionic pairs, we perorm sonography

every 4 weeks, starting at 16 to 20 weeks. Monochorionic twins

are imaged every 2 weeks or twin-twin transusion syndrome

(p. 848). o identiy suspected etal-growth restriction, we use

the standards o birthweight or twin gestations stratied by

placental chorionicity (Ananth, 1998).

■ Hypertension

Compared with mothers o singletons, those with multietal

gestations are more likely to develop a pregnancy-associated

hypertensive disorder, and the incidence urther rises with

advancing etal number (Day, 2005). In one analysis, 14 percent o parturients with twins developed a hypertensive disorder

o pregnancy (Aviram, 2021). No specic zygosity coners a

greater rate o these disorders (Lučovnik, 2016). However, with

twins and gestational diabetes, the preeclampsia incidence is

increased (Dave, 2021). Similar to singleton pregnancies with

hypertensive disorders, etal-growth restriction is a potential

outcome (Proctor, 2019) (Chap. 40, p. 702).

Data suggest that rising etal number and levels o antiangiogenic actor play roles in preeclampsia pathogenesis in multiples (Chap. 40, p. 694). Nonhypertensive women with twins

compared with those with singletons have higher levels o the

antiangiogenic actors soluble ms-like tyrosine kinase 1 (sFlt-1)

and soluble endoglin and lower levels o proangiogenic placental

growth actor (PlGF) (Faupel-Badger, 2015). In women with

twins and preeclampsia in one study, both sFlt-1 levels and

sFlt-1/PlGF ratios rose and PlGF concentrations declined compared with normotensive women with twins (Rana, 2012). Aspirin therapy is recommended to help prevent preeclampsia and

is discussed with prenatal care (p. 853).

■ Longterm Infant Development

In cohort studies evaluating twin and singletons, academic

perormance is similar and is unaected by AR-assisted conception (Christensen, 2006; Spangmose, 2017). For mono- or

dichorionic pairs born preterm, neurodevelopment at 2 years

was reported to be similar (osello, 2021).

However, among normal-birthweight neonates, the cerebral

palsy (CP) risk is twoold higher among multietal pregnancies compared with singletons. In term and preterm neonates

rom 12 large CP registries, the overall CP rate was 1.6 per

1000 singleton live births and 7 per 1000 live births rom multietal pregnancies (Perra, 2021). In this and another registry

study, the higher CP rate in multietal gestations was attributed mainly to prematurity (Sellier, 2021) (Chap. 34, p. 620).

Fetal-growth restriction, congenital anomalies, twin-twin trans-

usion syndrome, and etal demise o a co-twin are other potential contributors.

UNIQUE FETAL COMPLICATIONS

Several unique complications arise in multietal pregnancies.

Tese are described in twins but also can be ound in higherorder multietal gestations. Most etal complications attributed

to the twinning process itsel are seen with monozygotic twins.

Teir pathogenesis is best understood ater reviewing the possibilities shown in Figure 48-1.

■ Monoamnionic Twins

Tese twins make up less than 1 percent o all twin pregnancies

and 4 percent o monochorionic pairs (Chitrit, 2021; Sebire,

2000). However, they have high rates o many signicant complications.

Fetal loss rates in monoamnionic twins are substantial.

Among etuses alive beore 16 weeks’ gestation, less than hal

survive to the neonatal period. Fetal abnormalities and spontaneous miscarriage contribute to most losses (Preumo, 2015).

A high perinatal death rate is attributable to preterm birth,

twin-twin transusion syndrome, cord entanglement, birthweight discordance, and congenital anomalies (Buca, 2020;

Saccone, 2020).

Congenital anomaly rates in monoamnionic twins reach

18 to 28 percent (Post, 2015). Concordance o anomalies is

ound in only approximately one quarter o cases. Because o

the greater risk o cardiac anomalies, etal echocardiography

is indicated. O note, monoamnionic twins are by denition

monozygotic and thus presumed to be genetically identical,

except in rare cases (Zwijnenburg, 2010). Interestingly, the

risk or Down syndrome in each etus o the monozygotic pair

is similar to or lower than the risk in maternal age-matched

singletons (Sparks, 2016).

Twin-twin transusion syndrome rates in monoamnionic

twins are lower than in monochorionic diamnionic pregnancies

(Murgano, 2020). Tis may be due to the near universal presence in monoamnionic twins o arterioarterial anastomoses,

which are presumed to be protective (Hack, 2009). Nonetheless, twin-twin transusion syndrome surveillance is recommended and described in that section.

Umbilical cord entanglement is a requent event (Fig. 48-7).

Although color-ow Doppler sonography can help diagnose

entanglement, actors that lead to pathological umbilical vessel

constriction are unknown. Fetal death rom cord entanglement

is thus unpredictable, and monitoring or this is relatively ine-

ective. Moreover, studies o inpatient or outpatient etal surveillance o monoamnionic twin pregnancies show conicting

data. Heyborne and coworkers (2005) reported no stillbirths in

43 women admitted at 26 to 27 weeks’ gestation or daily etal

surveillance. Conversely, 13 etuses died in 44 women managed

as outpatients. In the MONOMONO Working Group (2019)

study o 195 women with these twins, the etal death rate was

3.3 percent in 75 mothers admitted at 24 to 29 weeks’ gestation compared with 10.8 percent in the 120 gravidas monitored outpatient. Notably, no etal deaths occurred ater 32

weeks’ gestation in either group. Last, Van Mieghem and associates (2014) ound the risk o “potentially preventable death”

was not signicantly dierent in women with inpatient (2.1

percent) or outpatient (4.7 percent) etal surveillance.

In the United States, mothers with monoamnionic twins are

oten admitted at 24 to 28 weeks’ gestation to begin 1 hour o

daily etal heart rate monitoring (American College o Obstetricians and Gynecologists, 2021b). Optimal surveillance is

unclear and may include nonstress testing or biophysical prole

assessment (Chap. 20, p. 386). Betamethasone, discussed later,

is considered to promote pulmonary maturation (p. 855). I

etal testing remains reassuring and no other intervening indications arise, cesarean delivery is perormed at 320/7 to 340/7

weeks’ gestation. o help manage these many care items, the

Society or Maternal-Fetal Medicine (2020) oers a prenatal

checklist.

■ Unique and Aberrant Twinning

Several aberrations in monozygotic twinning result in a spectrum o etal malormations. Tese are traditionally ascribed

to incomplete splitting o an embryo into two separate twins.

However, they theoretically may result rom early secondary

usion o two separate embryos. Tese separated embryos are

either symmetrical or asymmetrical. Te spectrum o asymmetrical twinning includes external parasitic twins, etus-in-etu,

and twin reversed-arterial-perusion (RAP) sequence, which

is described later (p. 850).

Conjoined Twins

Joining o the twins may begin at either pole and produce characteristic orms depending on which body parts are joined or

shared (Fig. 48-8) (Spencer, 2000). Te requency o conjoined

twins has a prevalence o 1.5 in 100,000 births, and thoracopagus is the most common type (Mutchinick, 2011).

Conjoined twins can be identied using sonography in

the rst trimester (Chen, 2011). Tis provides an opportunity or parents to decide whether to continue the pregnancy.

During sonographic interrogation, etal poles are seen to be

closely associated and do not change relative position rom one

another (Fig. 48-9). Other clues are more than three vessels

in the umbilical cord, ewer limbs than expected, spine hyper-

exion, bid etal pole, and increased nuchal thickness. Treedimensional ultrasound, color Doppler, and MR imaging are

valuable adjuncts to clariy shared organs (Baken, 2013).

Postnatal surgical separation may be successul i essential

organs are not shared. Conjoined twins may have discordant

structural anomalies that urther complicate decisions about

whether to continue the pregnancy. Consultation with a pediatric surgeon oten assists with parental decisions.

Viable-aged conjoined twins should be delivered by cesarean.

For pregnancy termination, however, vaginal delivery is possible

because the union is oten pliable. Still, dystocia is common,

and vaginal delivery may be traumatic to the uterus or cervix.

External Parasitic Twins and Fetus-in-fetu

Attached to a relatively normal twin, an external parasitic twin

is a grossly deective etus or merely etal parts. It usually consists o externally attached supernumerary limbs, oten with

some viscera. Classically, however, a unctional heart or brain

is absent. Parasitic twins are believed to derive rom a dead

deective twin, whose surviving tissue attaches to and receives

vascular support rom the normal co-twin (Spencer, 2001). In

one study, parasitic twins accounted or 4 percent o all conjoined twins (Mutchinick, 2011).

With etus-in-etu, one embryo may enold early within its

co-twin and mainly intraabdominally. Normal development

o this rare parasitic twin usually arrests in the rst trimester.

Tus, normal spatial arrangement and many organs are lacking.

Classically, vertebral or axial bones are ound in the etiorm

mass, whereas a heart and brain are absent. Tese masses are

thought to be a monozygotic, monochorionic diamnionic twin

gestation and are typically supported by large parasitic vessels

to the host (McNamara, 2016).

■ Monochorionic Twins and

Vascular Anastomoses

All monochorionic placentas likely share some anastomotic

connections. With rare exceptions, anastomoses between twins

are unique to monochorionic twin placentas. However, the

number, size, and direction o these seemingly haphazard connections vary markedly (Fig. 48-10). In one analysis o more

than 200 monochorionic placentas, the median number o

anastomoses was eight (Zhao, 2013).

Anastomoses may be artery-to-artery, vein-to-vein, or arteryto-vein communications and are located on the chorioni

surace o the placenta. In contrast to these supercial connections, deep artery-to-vein communications can extend through

the capillary bed o a given villus (Fig. 48-11). Tese deep arteriovenous anastomoses create a common villous compartment

or “third circulation” that has been identied in approximately

hal o monochorionic twin placentas.

Whether these anastomoses are dangerous to either twin

depends on the degree to which they are hemodynamically

balanced. In those with signicant pressure or ow gradients,

a shunt will develop between etuses. Tis chronic etoetal

transusion may result in several clinical syndromes that include

twin-twin transusion syndrome (TTTS), twin anemia–polycythemia sequence (TAPS), and twin reversed-arterial-perusion

(TRAP) sequence.

Twin-Twin Transfusion Syndrome

In this syndrome, blood is transused rom a donor twin to its

recipient sibling such that the donor may eventually become

anemic and its growth may be restricted. In contrast, the recipient becomes polycythemic and may develop circulatory overload

with heart ailure maniest as hydrops. Classically, the donor

twin is smaller and pale, and its recipient sibling is larger and

has volume excess. As a result, the recipient neonate may suer

hyperviscosity and occlusive complications. Polycythemia in the

recipient twin may also lead to severe hyperbilirubinemia and

kernicterus (Chap. 33, p. 606). S complicates 10 to 15

percent o monochorionic twins (Marwan, 2019).

Chronic S results rom unidirectional ow through

deep arteriovenous anastomoses. Deoxygenated blood rom a

donor placental artery is pumped into a cotyledon shared by

the recipient (see Fig. 48-11). Once oxygen exchange is completed in the chorionic villus, the oxygenated blood leaves the

cotyledon via a placental vein o the recipient twin. Unless compensated—typically through supercial arterioarterial anastomoses—this unidirectional ow leads to volume depletion in

the donor and volume excess in the recipient (Lewi, 2013).

S typically presents in midpregnancy when the donor

etus becomes oliguric rom hypovolemia and decreased

renal perusion (Society or Maternal-Fetal Medicine, 2013).

Tis etus develops oligohydramnios, and the recipient etus

develops severe hydramnios, presumably due to increased urine

production rom hypervolemia. Virtual absence o amnionic

uid in the donor sac prevents etal motion, giving rise to the

descriptive term stuck twin or polyhydramnios–oligohydramnios

syndrome—“poly–oli.” Tis amnionic uid imbalance is associated with growth restriction, contractures, and pulmonary

hypoplasia in the donor twin, and premature rupture o the

membranes and heart ailure in the recipient.

Fetal Brain Damage. Cerebral palsy, microcephaly, porencephaly, and multicystic encephalomalacia are serious complications associated with placental vascular anastomoses. Te exact

pathogenesis o neurological damage is not ully understood

but is likely caused by ischemic necrosis leading to cavitary

brain lesions. In the donor twin, ischemia results rom hypotension, anemia, or both. In the recipient, ischemia develops

rom blood pressure instability and episodes o proound hypotension (Lopriore, 2011). Cerebral lesions may also be associated with preterm delivery (Chap. 34, p. 618). In one review

o 315 liveborn etuses rom pregnancies with S, cerebral

abnormalities were ound in 8 percent (Quarello, 2007).

I one twin o an aected pregnancy dies, blood is acutely

transused rom high-pressure vessels o the living twin through

anastomoses to low-resistance vessels o the dead twin. Tis

leads rapidly to hypovolemia and possible ischemic antenatal

brain damage in the survivor (Fusi, 1990, 1991). A less likely

cause is emboli o thromboplastic material originating rom the

dead etus.

Te acuity o hypotension ollowing the death o one twin

with S makes successul intervention or the survivor nearly

impossible. Even with delivery immediately ater a co-twin

demise is recognized, the hypotension that occurs at the moment

o death has likely already caused irreversible brain damage

(Langer, 1997; Wada, 1998). As such, immediate delivery is not

considered benecial in the absence o another indication.

Diagnosis. S is diagnosed based on two sonographic criteria (Society or Maternal-Fetal Medicine, 2013). First, a monochorionic pregnancy is identied. Second, hydramnios dened

by a largest vertical pocket >8 cm in one sac and oligohydramnios dened by a largest vertical pocket <2 cm in the other

twin is ound. Growth discordance or growth restriction may

be ound with S, but these per se are not considered diagnostic criteria.

Organizations that include the American College o Obstetricians and Gynecologists (2021b), Society or Maternal–Fetal

Medicine (2013), and North American Fetal Terapy Network

recommend sonographic surveillance o pregnancies at risk or

S (Emery, 2015). Tese examinations begin at approximately 16 weeks’ gestation, and studies are perormed every 2

weeks. In one study o 108 monochorionic twin pairs, a sonographic evaluation interval >2 weeks was associated with a

higher Quintero stage at the time o S diagnosis ( Torson,

2011).

Once identied, S is typically classied by the Quintero (1999) staging system (Fig. 48-12):

• Stage I—discordant amnionic uid volumes as described in

the earlier paragraph, but urine is still visible sonographically

within the bladder o the donor twin

• Stage II—criteria of stage I, but urine is not visible within the

donor bladder

• Stage III—criteria of stage II and abnormal Doppler studies

o the umbilical artery, ductus venosus, or umbilical vein

• Stage IV—ascites or frank hydrops in either twin

• Stage V—demise of either fetus.

In addition to these criteria, cardiac unction o the recipient

twin also declines with S (Wohlmuth, 2018). Although

etal echocardiographic ndings are not part o the Quintero

staging system, many centers routinely perorm etal echocardiography or S. Teoretically, earlier diagnosis o etal

cardiomyopathy may identiy pregnancies that would benet

rom early intervention (Votava-Smith, 2015). One measure,

the myocardial perormance index (MPI), is a Doppler index o

each ventricle’s unction. Scoring systems that include cardiac

unction have been developed, but their useulness to predict

outcomes remains controversial (Miller, 2021b; Society or

Maternal-Fetal Medicine, 2013).

Management and Prognosis. Te prognosis or multietal gestations complicated by S is related to Quintero stage and

gestational age at presentation. Some stage I cases remain stable

or regress without intervention. However, 60 percent progress

(Emery, 2016; Stirnemann, 2021). Conversely, outcomes in

those identied at stage III or higher are much worse, and the

perinatal loss rate is 70 to 100 percent without intervention

(Miller, 2021b; Society or Maternal-Fetal Medicine, 2013).

Several therapies available or S include laser ablation o

vascular placental anastomoses, amnioreduction, and selective

eticide. Similar to amniocentesis (Chap. 17, p. 344), amnioreduction is needle drainage o excess amnionic uid.

Tese techniques have been evaluated in randomized trials.

Te Euroetus trial included 142 women with severe S

diagnosed beore 26 weeks’ gestation. Participants were randomly assigned to laser ablation o vascular anastomoses or to

serial amnioreduction (Senat, 2004). Laser ablation yielded a

76-percent rate o survival to age 6 months or at least one

twin compared with a 51-percent rate with amnioreduction.

Analyses o most randomized studies conrm better neonatal

outcomes with laser therapy compared with selective amnioreduction (Marwan, 2019; Roberts, 2008; Rossi, 2008, 2009).

However, evaluation o twins rom the Euroetus trial through

6 years o age did not demonstrate an additional survival benet beyond 6 months or improved neurological outcomes in

those treated with laser (Salomon, 2010). At this time, laser

ablation o anastomoses is preerred or severe S (stages

II–IV). Optimal therapy or stage I disease is controversial, and

laser ablation or expectant surveillance is an option (Emery,

2015; Stirnemann, 2021).

O techniques, an ablation method may laser individual

anastomoses or may ablate the entire the vascular equator. Tis

equator is the border between twin vasculature on the chorionic

surace. Equatorial ablation, which is reerred to as a Solomon

technique, reduces the likelihood o S recurrence. Tis

method also lowers rates o APS, which can be a laser-therapy

sequelae and described next (Slaghekke, 2014).

Ater laser therapy, close ongoing surveillance is necessary.

Weekly ultrasound and Doppler studies are recommended

(Marwan, 2019; Miller 2021b). Tese evaluations monitor

etal growth, amnionic uid volumes, placental unction, and

anemia. Delivery timing is usually inuenced by S recurrence, etal-growth restriction, or by abnormal Doppler velocimetry values, which reect poor placental unction (Chap. 47,

p. 830).

Another treatment option, selective etal reduction, has generally been considered i severe amnionic uid and growth disturbances develop beore 20 weeks. In such cases, both etuses

typically will die without intervention. Any substance injected

into one twin may aect the other twin because o shared circulations. Tus, or the etus chosen or reduction, eticidal

methods aim to occlude the umbilical vein or umbilical cord.

Radiorequency ablation, etoscopic ligation, or coagulation

with laser, monopolar, or bipolar energy are options (Challis,

1999; Chang, 2009; Parra-Cordero, 2016). Even ater these

procedures, however, risks to the remaining etus are still appreciable (Rossi, 2009). Early termination o the entire pregnancy

is yet another option.

Twin Anemia–Polycythemia Sequence

Chronic etoetal transusion underlies this orm, which is

characterized by signicant hemoglobin dierences between

donor and recipient twins. Similar to S, the donor twin

in spontaneous APS is anemic and usually smaller than the

recipient twin, which is polycythemic. However, APS lacks

the discrepancies in amnionic uid volumes typical o S.

Spontaneous APS can develop at any gestational age and

complicates 1 to 6 percent o monochorionic twins (Marwan,

2019). Iatrogenic APS develops in up to 13 percent o pregnancies ater laser ablation o the placenta and usually develops

within 5 weeks o a procedure (Lewi, 2013; ollenaar, 2021).

In iatrogenic APS, the ormer S recipient twin usually

becomes anemic, whereas the ormer donor becomes polycythemic (ollenaar, 2016).

Sonographically measuring blood ow velocity in the etal

middle cerebral artery (MCA) can accurately identiy etal

anemia (Chap. 14, 262). Antenatally, APS is diagnosed by

discordant MCA peak systolic velocity (PSV) values between

twins. Specically, an MCA-PSV value that is >1.5 multiples

o the median (MoM) in the donor twin and <1.0 MoM in

the recipient twin (Society or Maternal-Fetal Medicine, 2013).

Others have suggested alternative threshold values (Khalil,

2020; ollenaar, 2019).

A staging system or APS has been proposed, and higher

stage is associated with increased perinatal mortality rate

( Slaghekke, 2010; ollenaar, 2021). Screening or spontaneous APS is controversial, as improvements in perinatal outcomes have not been demonstrated (Khalil, 2016; Society or

Maternal-Fetal Medicine, 2013).

Management options include expectant care, delivery, laser

surgery, intrauterine transusion, selective eticide, and pregnancy termination. A clinical trial evaluating etoscopic laser

therapy or APS is currently ongoing. Antenatal surveillance

mirrors that just described or S. Again, delivery timing is

usually inuenced by worsening etal growth or by abnormal

Doppler velocimetry values. Postnatal treatment oten requires

blood transusion or the donor twin and partial exchange

transusion o the recipient twin.

Twin Reversed-arterial-perfusion Sequence

Also known as acardiac twinning, this rare, serious complication o monochorionic multietal gestation has an estimated

incidence o 1 case in 35,000 births. With classic RAP

sequence, the twin pair is a normally ormed donor twin that

shows eatures o heart ailure and a grossly malormed recipient twin that lacks a heart (acardius) and other structures. In

one theory, the RAP sequence is caused by a large artery-toartery placental shunt, oten also accompanied by a vein-tovein shunt (Fig. 48-13). Within the single, shared placenta,

arterial perusion pressure o the donor twin exceeds that in the

recipient twin. Te recipient thus receives reversed blood ow

containing deoxygenated arterial blood rom its co-twin (Lewi,

2013). Tis “used” arterial blood reaches the recipient twin

through its umbilical arteries and preerentially goes to its iliac

vessels. Tus, only the lower body is perused, and disrupted

growth and development o the upper body ollows. In these

cases, ailed head growth is called acardius acephalus; a partially

developed head with identiable limbs is called acardius myelacephalus; and ailure o any recognizable structure to orm is

acardius amorphous, which is shown in Figure 48-14. Because

o this vascular connection, the normal donor twin must not

only support its own circulation but also must pump blood to

and through the underdeveloped acardiac recipient. Tis may

lead to cardiomegaly and high-output heart ailure in the donor

twin (Fox, 2007; Marwan, 2019).

In the past, the donor-twin mortality rate exceeded 50 percent. Tis stemmed largely rom complications o prematurity

or rom a prolonged high-output state leading to cardiac ailure

(Dashe, 2001). Risk is directly related to size o the acardiac

twin. When the acardiac twin is large, treatment is generally

oered. Radiorequency ablation (RFA) is the preerred modality o therapy, and contemporary reports now suggest improved

perinatal outcomes. Te North American Fetal Terapy Network reviewed their experiences with 98 cases rom 1998 to

2008 in which RFA o the umbilical cord was perormed.

Median gestational age at delivery was 37 weeks’ gestation, and

80 percent o donor neonates survived (Lee, 2013).

■ Hydatidiform Mole with Coexisting

Normal Fetus

Tis rare gestation contains one normal etus, and its co-twin

is a complete molar pregnancy. It must be dierentiated rom

a partial molar pregnancy, which is a singleton, triploid etus

and its placenta composed o molar tissue (Fig. 13-4, p. 238).

Diagnosis in the rst hal o pregnancy is common. Sonographically, a normal-appearing twin is accompanied by its

co-twin, which is a large placenta containing multiple small

anechoic cysts. Oten, these pregnancies are terminated, but

pregnancy continuation is increasingly adopted. Te live birth

rate rom one review was 50 percent (Zilberman, 2020). Te

risk o gestational trophoblastic neoplasia (GN), which is a

malignant sequelae o hydatidiorm mole, is similar whether

the pregnancy is terminated or not (Massardier, 2009; Sebire,

2002). Given the limited number o cases, robust data or

rm recommendations are lacking. Discussed in Chapter 13

(p. 237), complications o expectant management include

vaginal bleeding, hyperemesis gravidarum, thyrotoxicosis, and

early-onset preeclampsia. Many o these complications result

in preterm birth with its attendant adverse perinatal outcomes.

Logically, close antepartum and postpartum surveillance is

needed or those continuing the pregnancy. Postpartum GN

surveillance is essential and described in Chapter 13 (p. 240).

DISCORDANT GROWTH

Fetal size inequality develops in approximately 15 percent o

twin gestations (Miller, 2012). Generally, as the weight dierence within a twin pair grows, the perinatal mortality rate rises

proportionately. Earlier discordancy and monochorionicity

pose increased mortality risks or the smaller twin. Specically,

with discordant growth identied at or beore 20 weeks’ gestation in studies, 8 to 15 percent o the growth-restricted etuses

die (Couck, 2020; Curado, 2020; D’Antonio, 2018).

■ Pathogenesis

Te etiology o growth discordance in monochorionic

twins likely diers rom that in dichorionic twins. First, in

monochorionic twins, the single placenta is not always equally

shared, and this leads to higher discordant growth rates than

in dichorionic pairs (Fig. 48-15). In cases with S, discordancy in monochorionic twins is usually attributed to placental

anastomoses that cause a perusion imbalance between twins.

Reduced perusion o the donor twin can diminish placental

and etal growth (Lewi, 2013). Last, monochorionic twins at

times can be discordant in size because they are discordant or

structural anomalies.

In dichorionic twins, discordancy may result rom various

actors. Dizygotic etuses may have dierent genetic growth

potential, especially i they are o opposite genders. Second,

because the placentas are separate and require more implantation space, one placenta might have a suboptimal implantation

site. Additionally, umbilical cord abnormalities such as velamentous insertion, marginal insertion, or vasa previa may play

a role (Chap. 6, p. 114). One study showed that the incidence

o severe discordancy is twice as great in triplets as it is in twins

(Bagchi, 2006). Tis supports the view that in-utero crowding promotes multietal growth restriction. Placental pathology

may be contributory. In one study o 668 twin placentas, a

strong relationship between histological placental abnormalities

and birthweight discordancy was observed in dichorionic but

not monochorionic twin pregnancies (Kent, 2012).

■ Diagnosis

Antenatal size discordancy between twins can be best determined sonographically. Crown-rump length dierences are not

reliable predictors or birthweight discordance (Miller, 2012).

Tus, most discordancy surveillance begins ater the rst trimester. One common method uses sonographic etal biometry

to compute an estimated weight or each twin (Chap. 15, p.

274). Percent discordancy is then calculated as the weight o

the larger twin minus the weight o the smaller twin, divided by

the weight o the larger twin. Te American College o Obstetricians and Gynecologists (2021b) denes discordance as an

estimated etal weight dierence >20 percent.

■ Management

Te risk or adverse perinatal outcomes in the setting o growth

discordance remains controversial. Some suggest that twin gestations with discordant growth between two etuses that are

appropriately grown or gestational age are not at increased risk

or adverse outcomes (American College o Obstetricians and

Gynecologists, 2021b; Appleton, 2007). Others have shown an

increased risk or adverse etal outcomes but not or poor neonatal outcomes (Amaru, 2004; D’Antonio, 2018). However,

accumulated data suggest that weight discordancy exceeding

25 to 30 percent most accurately predicts an adverse perinatal outcome (Cohen, 2001; Chen, 2019). At Parkland Hospital, twin weight-discordancy values rom 1370 delivered twin

pairs were stratied by percentage increments (Hollier, 1999).

Te incidence o respiratory distress syndrome, intraventricular hemorrhage, seizures, periventricular leukomalacia, sepsis,

and necrotizing enterocolitis rose directly with the percentage

o weight discordancy. Rates o these conditions grew substantially i discordancy exceeded 25 percent. Te relative risk o

etal death increased signicantly to 5.6 i weight discordancy

was >30 percent and rose to 18.9 i >40 percent.

Nonstress testing and biophysical prole assessment have

all been recommended in management o twin growth discordancy. I signicant discordancy is identied in a monochorionic twin pair, umbilical artery Doppler studies in the smaller

etus may help guide management (Gratacós, 2007). Data are

limited to establish the optimal timing o delivery o twins with

size discordancy alone. At advanced gestational ages, delivery

can be pursued.

Selective Fetal-growth Restriction

Restricted growth o one twin etus is termed selective etalgrowth restriction (sFGR) and usually develops late in the second

and early third trimester. Some diagnose sFGR i the abdominal

circumerence (AC) measurement dierence exceeds 20 mm or

i etal-growth discordance is >20 percent (Khalil, 2019). I

sFGR is diagnosed, weekly testing o etal well-being, evaluation o amnionic uid volume, and umbilical artery Doppler

velocimetry are undertaken. Investigators have correlated Doppler results with placental ndings and with the degree o sFGR

to predict etal outcome (Gratacós, 2012). Tese correlations

have yielded categories o sFGR.

• Type I shows positive end-diastolic ow, a smaller degree of

weight discordance, and a relatively benign clinical course.

• Type II displays persistently absent end-diastolic ow in the

smaller twin and carries a high risk o deterioration and demise.

• Type III has intermittently absent or reversed end-diastolic

ow. Because o large artery-to-artery anastomoses associated

with the placentas in this last category, type III is associated

with a lower risk o deterioration than type II. In all evaluated

cases, unequally shared placenta was noted to some degree.

Serial evaluation o etal growth is perormed every 3 weeks

(Chap 47, p. 829). At Parkland Hospital, daily inpatient surveillance is undertaken in women with etal-growth restriction

in one twin or with twin discordancy exceeding 25 percent in

the setting o a twin with restricted growth.

FETAL DEMISE

At any time during multietal pregnancy, one or more etuses

may die, either simultaneously or sequentially. Most causes

stem rom complications o etal anomaly or chorionicity.

Related to the latter, compared with dichorionic twins, monochorionic pairs suer higher rates o sFGR, S, or APS

rom unequal vascular anastomoses, and monoamnionic pairs

can die rom cord entanglement.

When death is early, it may maniest as a vanishing twin (p.

843). For the survivor, the risk o death ater the rst trimester

is not increased, and the pregnancy requires no additional surveillance or this specic indication. In a slightly more advanced

gestation, the dead etus may be compressed appreciably—etus

compressus, or it may be attened remarkably through desiccation—

etus papyraceus (Fig. 48-16).

In gestations past 20 weeks, the stillbirth rate in twins in the

United States was 1.4 percent o live births and exceeded the

0.6-percent rate in singletons (MacDorman, 2015). Ater a single

intrauterine etal demise (sIUFD) in these later gestations, risks to

the remaining co-twin include death, preterm birth, and neurological injury (D’Antonio, 2017; Mackie, 2019). With the last, acute

hypovolemia rom volume shits within placental anastomoses

occurs immediately, and neurological injury is not preventable.

Related to co-twin death, monochorionic diamnionic twins

with an sIUFD were 16 times more likely to experience death

o the co-twin than were dichorionic twins with an sIUFD in

one large series (Morikawa, 2012). In most studies, the rate

o co-twin demise ater sIUFD declines ater 26 to 28 weeks’

gestation and with advancing gestational age (Mackie, 2019;

McPherson, 2012; Southwest Tames Obstetric Research

Collaborative, 2012; Wood, 2014).

Te rate o preterm birth ollowing sIUFD is increased but

is similar between mono- and dichorionic twins. Advancing

rates are seen at later gestational ages (D’Antonio, 2017; Ong,

2006).

Te neurological prognosis or a surviving co-twin is inuenced by chorionicity. In one review o diamnionic twin

pregnancies complicated by sIUFD beore 34 weeks, neurodevelopmental morbidity was 29 percent in monochorionic

twins and was threeold higher than in dichorionic gestations

(Mackie, 2019). In other series with sIUFD ater 34 weeks,

the likelihood o neurological decits was essentially the same

between monochorionic and dichorionic twin pregnancies

(Hillman, 2011).

Because o this signicant neurological risk, i one etus o

a monochorionic twin gestation dies ater the rst trimester

but beore viability, pregnancy termination can be considered

(Blickstein, 2013). Ater viability, ultimate delivery timing

balances the risk o prematurity against co-twin demise. Early

management emphasizes maternal and co-twin saety and gestation prolongation (American College o Obstetricians and

Gynecologists, 2021b). Antenatal corticosteroids or survivor

lung maturity can be considered. For sIUFD occurring ater 34

weeks’ gestation, delivery is reasonable (Spong, 2011).

Last, the death o one etus could theoretically trigger coagulation deects in the mother. Only a ew cases o maternal coagulopathy ater a single etal death in a twin pregnancy have been

reported. Tis is probably because the surviving twin is usually

delivered within a ew weeks o the demise (Eddib, 2006).

PRENATAL CARE

At Parkland Hospital clinics, women with multietal gestations

are seen every 2 weeks beginning at 22 weeks’ gestation. One

imperative is preterm delivery prevention, and a digital cervical examination is perormed at each visit to screen or cervical

shortening or dilation.

Te maternal diet should provide additional requirements

or calories, protein, minerals, vitamins, and essential atty acids.

Te Institute o Medicine (IOM) (2009) recommends a 37- to

54-lb weight gain or women with twins and a normal body

mass index. Te daily recommended augmented caloric intake

or women with twins is 40 to 45 kcal/kg/d. Diets contain 20

percent protein, 40 percent carbohydrate, and 40 percent at

divided into three meals and three snacks daily. Based on upperintake levels rom the Food and Nutrition Board o the IOM,

one review endorsed supplementation o calcium, magnesium,

zinc, and vitamins C, D, and E (Goodnight, 2009).

Discussed in Chapter 40 (p. 705), low-dose aspirin prophylaxis is recommended in women at high risk o preeclampsia,

which includes those with a multietal pregnancy (LeFevre,

2014). An 81-mg oral daily dose is initiated between 12 and

28 weeks’ gestation and is continued until delivery (American

College o Obstetricians and Gynecologists, 2020b).

Prenatal screening or etal aneuploidy is carried out as

described in Chapter 17 (p. 334). Tus, the combined test

or secondary screening is done and interpreted with value

thresholds set or multietal pregnancies. Noninvasive prenatal

screening with cDNA also is acceptable (American College o

Obstetricians and Gynecologists, 2020d; Judah, 2021; Khalil,

2021). Interpretation caveats or those with a vanishing twin

were described earlier (p. 844).

o screen or structural anomalies, a midpregnancy anatomic sonography survey is perormed. In addition, monochorionic twins undergo echocardiography because o an increased

risk or cardiac anomalies (Bahtiyar, 2007). As noted earlier

(p. 844), etal growth is assessed with sonography. Amnionic

uid volume is quantied using the deepest vertical pocket in

each sac. A measurement <2 cm is considered oligohydramnios, and a measurement >8 cm is considered hydramnios

(Duryea, 2017; Hernandez, 2012).

win pregnancies are at increased risk or stillbirth compared to singleton gestations. Te intrauterine etal death rate

is two- to threeold higher in monochorionic pregnancies

compared with dichorionic gestations (Lee, 2008; Morikawa,

2012). Cheong-See and colleagues (2016) reported a higher

stillbirth rate in monochorionic gestations compared with

dichorionic gestations beyond 34 weeks’ gestation. Outpatient

weekly nonstress test or biophysical prole may help lower the

rate o intrauterine death in twin gestations (Booker, 2015;

Burgess, 2014). Weekly antenatal surveillance is undertaken at

36 weeks’ gestation or uncomplicated dichorionic pregnancies

and at 32 weeks’ gestation or uncomplicated monochorionic

gestations (American College o Obstetricians and Gynecologists, 2021a). However, all antenatal testing schemes have high

alse-positive rates. In cases o abnormal testing in one twin and

normal results in another, iatrogenic preterm delivery remains a

major concern. Tereore, patients are careully counseled prior

to initiating antenatal testing in twins.

PRETERM BIRTH

Gestational length shortens and preterm birth risk rises with

accruing etal number. In 2019 in the United States, the

preterm birth rate in twins and triplets was 60 and 98 percent,

respectively (Martin, 2021). One review showed that approximately 60 percent o preterm births in twins are indicated, a

third result rom spontaneous labor, and 10 percent ollow

preterm prelabor rupture o membranes (PPROM) (Chauhan,

2010).

O risks, preterm birth rates vary with chorionicity and are

higher with monochorionic compared with dichorionic twins.

Other implicated actors are prior preterm birth, adolescence,

nulliparity, obesity, and diabetes (Marleen, 2018, 2021).

■ Prediction

One goal o multietal prenatal care is to identiy women likely

to experience preterm delivery. o and associates (2006) sonographically measured cervical length in 1163 twin pregnancies

at 22 to 24 weeks’ gestation. Rates o preterm delivery beore

32 weeks were 66 percent in those with cervical lengths o 10

mm; 24 percent or lengths o 20 mm; and only 1 percent or

lengths o 40 mm. In one review, a cervical length <20 mm

was most accurate or predicting birth beore 34 weeks’ gestation. Te specicity was 97 percent, and positive likelihood

ratio was 9.0 (Conde-Agudelo, 2010). Notably, a closed

internal os by digital examination predicted postponed delivery

equally well as the combination o a normal sonographically

measured cervical length and negative etal bronectin test

result ( McMahon, 2002).

Unortunately, cervical length assessment in twin pregnancies has not led to improved outcomes (Gordon, 2016). Te

Society or Maternal–Fetal Medicine (2016) recommends

against routine cervical length screening in multietal gestations.

Fetal bronectin levels also may predict preterm birth (Marleen, 2020). Again, neonatal outcomes are not improved in

twins, and this screening tool is not recommended (American

College o Obstetricians and Gynecologists, 2021b).

■ Prevention

Bed Rest

In general, most strategies or preterm birth prevention are

ineective or singleton and multietal pregnancies ( American

College o Obstetricians and Gynecologists, 2021b). O

options, bed rest with or without hospitalization does not

prolong multietal pregnancy. In one metaanalysis, the practice did not reduce the risk o preterm birth (da Silva Lopes,

2017). At Parkland Hospital, elective hospitalization was

compared with outpatient management, and no advantages

were ound (Andrews, 1991). Importantly, however, almost

hal o women managed as outpatients required admission or

specic indications such as hypertension or threatened preterm delivery.

Limited physical activity, early work leave, more requent

health-care visits, serial sonographic examinations, and structured maternal education regarding preterm delivery risks have

been advocated to reduce preterm birth rates in women with

multiple etuses. However, little evidence suggests that these

practices substantially change this outcome.

Prophylactic Tocolysis

Tis has not been studied extensively in multietal pregnancies.

In one review o prophylactic oral beta-mimetic therapy that

included 374 twin pregnancies, treatment did not reduce the

rate o twins delivering beore 37 or beore 34 weeks’ gestation

(Yamasmit, 2015). In light o the Food and Drug Administration warning against the use o oral terbutaline because o

maternal side eects, the prophylactic use o beta-mimetic

drugs in multietal gestations seems unwarranted.

Progesterone Therapy

17-alpha-hydroxyprogesterone caproate (17-OHPC) injections are

not eective or multietal gestations (Caritis, 2009; Combs,

2011; Rouse, 2007). Moreover, women carrying twins and

having a cervical length <36 mm or a length <25 mm did not

benet despite their greater risk or preterm birth (Durnwald,

2010; Senat, 2013).

Vaginal micronized progesterone in various ormulations in

randomized studies has also proved ineective or preterm

birth prevention beore 34 weeks in a general twin population

(Norman, 2009; Rode, 2011). For women with a short cervix,

subgroup analysis and a metaanalysis show conicting results

regarding benets (D’Antonio, 2021; Klein, 2011; Rehal,

2021). At Parkland Hospital, current management o multietal

gestations does not typically include any progesterone therapy.

Cervical Cerclage

Prophylactic cerclage does not improve perinatal outcome in

women with twin pregnancies. Studies have included women

with and without a short cervix (D’Antonio, 2021; Jarde, 2017;

Newman, 2002). Physical examination–indicated cerclage in

women with a second-trimester twin gestation and a dilated

cervix may be benecial. Roman and coworkers (2016, 2020)

reported that women undergoing rescue cerclage plus prophylactic antibiotic and indomethacin administration beore 24

weeks’ gestation had lower preterm birth and perinatal mortality rates than those without a cerclage.

Pessary

Te silicone Arabin vaginal pessary encircles and theoretically

compresses the cervix, alters the inclination o the cervical

canal, and relieves direct pressure on the internal cervical os.

In a study o its use in women with a short cervix between 18

and 22 weeks’ gestation, a subgroup analysis o 23 women with

twins showed a signicant reduction in the delivery rate beore

32 weeks compared with the rate in 23 control pregnancies

(Arabin, 2003). In another randomized trial o twins, women

treated with a cervical pessary had signicantly ewer births

beore 34 weeks (Goya, 2016).

Other randomized studies comparing pessary against expectant care are less avorable. In one trial with 1180 uncomplicated twin pregnancies, pessary use ailed to alter the preterm

birth rate (Nicolaides, 2016). Another trial o 813 unselected

women had similar ndings. However, delivery rates beore

32 weeks were lower—29 versus 14 percent—in a subset o

women with a cervical length <38 mm (Liem, 2013). Instead,

other trials show no benet rom pessary use in this subgroup

(Nicolaides, 2016; Norman, 2021). At this time, pessary use is

not recommended by the American College o Obstetricians

and Gynecologists (2021b).

■ Treatment

Tocolytic therapy to help halt preterm labor in multietal

pregnancy does not measurably improve neonatal outcomes

( Canadian Preterm Labor Investigators Group, 1992; Yamasmit,

2015). Moreover, tocolytic therapy in women with a multietal

pregnancy entails higher maternal risk than in singleton pregnancy. Tis stems in part rom augmented pregnancy-induced

hypervolemia, which raises cardiac demands and increases the

susceptibility to iatrogenic pulmonary edema (Chap. 50, p.

883). In one study, all women with a multietal gestation treated

with a beta-mimetic drug or preterm labor had more cardiovascular complications—43 versus 4 percent—than women with

singletons (Gabriel, 1994). In a retrospective analysis, niedipine

tocolysis in 58 singleton and 32 twin pregnancies led to higher

incidences o side eects such as maternal tachycardia in women

with twins—19 versus 9 percent (Derbent, 2011).

Antenatal corticosteroids or etal lung maturation have

not been well studied in multietal gestations. O studies,

many compare outcomes among singletons and twins whose

mothers either did or did not receive this therapy. In one

large retrospective study evaluating betamethasone, neonatal morbidity rates were reduced in both twin and singleton

treated groups (Melamed, 2016). Another similar comparison ound reduced rates o periventricular hemorrhage rates

in treated groups but not improved respiratory distress syndrome (RDS) rates (Gonçalves-Ferri, 2021). Similarly, in

another study, short-term neurological outcome was better in

treated twin and singletons, but rates o mortality, RDS, and

cerebral palsy were not reduced (Ushida, 2020). However, in

one study solely with twins, the rate o RDS or o composite

neonatal morbidity was not lower with corticosteroid therapy

(Viteri, 2016). Tese authors posited pharmacokinetics in

twin gestation as a potential explanation. Currently, recommended use o agents is the same or multietal and singleton

gestations (American College o Obstetricians and Gynecologists, 2021b). Tis therapy is described ully in Chapter 45

(p. 802).

■ Preterm Prelabor Membrane Rupture

Te requency o PPROM rises with increasing plurality. In

a population-based study o more than 290,000 live births,

the proportion o preterm birth complicated by PPROM was

13 percent in singletons. Tis rate was 17, 20, 20, and 100

percent in twins, triplets, quadruplets, and higher-order multiples, respectively (Pakrashi, 2013). For both twin and singleton pregnancies, most studies show comparable neonatal risk

with expectant management, which is outlined in Chapter 45

(p. 798) (Kibel, 2017). However, the time between births,

oten termed latency, is shorter with twins. With PPROM

ater 24 weeks’ gestation, the median number o days to subsequent delivery was 4 days or twins compared with 7 days

or singletons in one study (Madden, 2021). Ater 30 weeks’

gestation, this latency or twins is signicantly shortened

(Mercer, 1993).

■ Delayed Delivery of Second Twin

Rarely, ater preterm birth o one etus, it may be advantageous or undelivered etus(es) to remain in utero. Tis may

be especially so at periviable gestational ages. However, this

advantage must be balanced against substantial maternal risk.

In one systematic review, nearly 40 percent o mothers su-

ered complications with delayed delivery (Cheung, 2020).

Tese include inection, postpartum hemorrhage, and placental abruption.

From population-based data o delayed deliveries, the

median latency duration was 6 days (range 6 to 107 days) in

200 twin pregnancies (Zhang, 2004). In all studies, retained

etuses had better overall survival rates than the rst-born neonate. In one series o 38 twins, the mean latency was 19 days.

O rst-born twins born at <25 weeks’ gestation, none survived, but 50 percent o their co-twins did. Beyond 25 weeks,

survival rates were 65 or rst-born and 95 percent or secondborn twins (Arabin, 2009). In their series o 28 twin pairs,

cerclage, antibiotic therapy, and tocolysis did not improve

outcomes (Fayad 2003).

I delayed delivery is attempted, counseling should include

the potential or serious maternal complications. Te range

o gestational age or which benets outweigh the risks or

delayed delivery is likely narrow, and gestations o 22 to 24

weeks would seem the most probable to benet (Oyelese,

2005). In our experience, good candidates or delayed delivery

are rare.

LABOR AND DELIVERY

■ Delivery Timing

Several actors aect this timing and include gestational age,

etal growth, maternal complications, lung maturity, and stillbirth risk. Te substantially greater stillbirth rate in monochorionic monoamnionic twins was discussed earlier (p. 845).

In dichorionic diamnionic twins, stillbirth rates rose to 10.6

deaths per 1000 pregnancies at 386/7 weeks’ gestation in a systematic review o more than 30,000 twin pairs. Te peak rate

in monochorionic diamnionic twins was 9.6 deaths per 1000

pregnancies at 376/7 weeks (Cheong-See, 2016).

In uncomplicated twins, stillbirth risk is balanced against

the morbidity o prematurity. Pulmonary maturation is usually

synchronous in twins and occurs several weeks earlier than in

singletons (Leveno, 1984). However, in some cases, pulmonary

unction may dier. With sFGR, the smaller, stressed twin has

lower rates o RDS but higher rates o bronchopulmonary dysplasia (Groene, 2021).

Te American College o Obstetricians and Gynecologists (2021b) recommends delivery at 380/7 to 386/7 weeks’

gestation or uncomplicated dichorionic diamnionic twin

pregnancies. Uncomplicated monochorionic diamnionic

twin pregnancies can be delivered between 340/7 and 376/7

weeks. For monochorionic monoamnionic twin pregnancies,

delivery is recommended at 320/7 to 340/7 weeks. At Parkland

Hospital, we generally ollow these recommendations and do

not routinely deliver monochorionic diamnionic twin pregnancies beore 37 weeks unless another obstetrical indication

develops.

■ Preparations

A litany o complications may be encountered during labor and

delivery o multiple etuses. Rates o uterine contractile dys-

unction, abnormal etal presentation, umbilical cord prolapse,

placenta previa, placental abruption, emergent operative delivery, and postpartum hemorrhage rates rom uterine atony are

higher. Moreover, second twins at term have worse composite

neonatal outcomes compared with outcomes o their co-twin

regardless o delivery method (Smith, 2007; Torngren-Jerneck,

2001). All o these must be anticipated, and thus certain precautions and special arrangements are prudent. Tese include

the ollowing:

1. An appropriately trained obstetrical attendant should

remain with the mother throughout labor. Continuous

electronic monitoring is preerable. I membranes are ruptured and the cervix dilated, the presenting etus is monitored internally

2. An intravenous inusion system capable o delivering uid

rapidly is established. In the absence o hemorrhage, lactated

Ringer or an aqueous dextrose solution is inused at a rate o

60 to 125 mL/hr.

3. Blood or transusion is readily available i needed.

4. An obstetrician skilled in intrauterine identication o etal

parts and in intrauterine manipulation o a etus should be

present.

5. A sonography machine is readily available to evaluate the

presentation and position o the etuses during labor and to

image the remaining etus ater delivery o the rst.

6. An anesthesia team is immediately available in the event that

emergent cesarean delivery is necessary or that intrauterine

manipulation is required or vaginal delivery.

7. For each etus, at least one attendant who is skilled in resuscitation and care o newborns and who has been appropriately inormed o the case should be immediately available.

8. Te delivery area should provide adequate space or the

nursing, obstetrical, anesthesia, and pediatric team members

to work eectively. Equipment must be on site to provide

emergent anesthesia, operative intervention, and maternal

and neonatal resuscitation.

■ Analgesia and Anesthesia

For multietal pregnancies, decisions regarding analgesia and

anesthesia must actor planned route o delivery. Other potential problems may stem rom preterm labor, preeclampsia,

desultory labor, need or intrauterine manipulation, and postpartum uterine atony and hemorrhage.

Labor epidural analgesia is ideal because it provides excellent pain relie and can be rapidly extended cephalad i internal podalic version or cesarean delivery is required. I general

anesthesia becomes necessary or intrauterine manipulation

during vaginal birth, uterine relaxation can be accomplished

rapidly with a halogenated inhalation agent (Chap. 25, p.

481). Some clinicians use intravenous or sublingual nitroglycerin or intravenous terbutaline to achieve uterine relaxation. I used, these agents are usually best administered by

the anesthesia team.

■ Fetal Presentation

For labor and delivery, the etal presentations are best determined sonographically. I active labor is conrmed, the decision or vaginal or cesarean delivery is reached, and etal

presentation is a major actor. Among the possible presentation

combinations, those most common at admission or delivery

are cephalic-cephalic, cephalic-breech, and cephalic-transverse.

At Parkland Hospital between 2008 and 2013, 71 percent o

twin pregnancies had a cephalic presentation o the rst etus

at the time o labor and delivery admission. During parturition, etal presentation o a second twin can be unstable. For

them, compound or ootling breech presentations and ace

or brow attitudes are relatively common and even more so i

etuses are small or numerous, amnionic uid is excessive, or

maternal parity is high. Cord prolapse also is requent in these

circumstances.

■ Delivery Route

Cephalic First Twin

With cephalic–cephalic presentation, general consensus supports

consideration o vaginal birth in a laboring woman near term

(American College o Obstetricians and Gynecologists, 2021b;

D’Alton, 2010). From studies, planned cesarean delivery does

not improve neonatal outcome when both twins are cephalic

(de Castro, 2016; Schmitz, 2017). With trials o labor, vaginal

delivery rates approximate 80 percent.

With cephalic–noncephalic presentation, the optimal delivery route remains controversial. Patient selection and provider

expertise with vaginal breech delivery is crucial and described in

Chapter 28 (p. 527). As a result, one common option is cesarean delivery o both twins. Less oten, ater spontaneous vaginal

delivery o a rst twin, intrapartum external cephalic version o

the second twin can be perormed. In case series, this practice,

compared with internal podalic version, was associated with

higher rates o intrapartum cesarean delivery and etal distress

(Gocke, 1989; Smith 1997). With internal podalic version, a

hand placed into the uterus grasps etal eet to deliver the etus by

breech extraction (Fig. 48-17). Last and least desirable is vaginal

delivery o the rst but cesarean delivery o the second twin due

to intrapartum complications. Tese are umbilical cord prolapse,

placental abruption, contracting cervix, or nonreassuring etal

heart rate. Most but not all studies report the worst composite

etal outcomes or this scenario (Alexander, 2008; Rossi, 2011).

For cephalic–noncephalic twins, each with birthweights

>1500 g and gestational ages >32 weeks, several reports attest

to the saety o vaginal delivery. In one study o 5915 pregnancies with a cephalic rst twin and either cephalic or noncephalic

second twin, 25 percent planned or cesarean delivery (Schmitz,

2017). Te other 75 percent planned a trial o vaginal delivery, which was successul in 80 percent. Interestingly, perinatal

mortality and morbidity rates were signicantly higher in the

planned cesarean delivery group delivered at <37 weeks—

5.2 versus 3.0 percent, respectively. Additionally, or those

delivered vaginally, neonatal outcomes were similar or second

twins that were cephalic or noncephalic (Schmitz, 2018). Te

win Birth Study had similar inclusion criteria and randomly

assigned women to planned vaginal or planned cesarean delivery. Perinatal outcomes were similar in both groups. For those

delivered vaginally, neonatal outcomes were similar or second

twins that were cephalic or noncephalic (Barrett, 2013).

For neonates weighing <1500 g compared with those weighing more, comparable or even better etal outcomes with vaginal delivery compared with cesarean delivery have been reported

(Mol, 2020; Sentilhes, 2015). However, rates o urgent operations or the second twin may be higher (Hiersch, 2021).

Other investigators advocate cesarean delivery or both

etuses o a cephalic–noncephalic twin pair (Armson, 2006;

Homann, 2012). Yang and coworkers (2005a,b) studied

15,185 cephalic–noncephalic pairs. Te risks o asphyxiarelated neonatal deaths and morbidity were higher in the group

in which both twins were delivered vaginally compared with

the group in which both twins were delivered surgically.

Breech First Twin

Problems with the rst twin presenting as a breech are similar to

those encountered with a singleton breech etus. First, the etal

body can be small, and delivery o the extremities and trunk

through an inadequately eaced and dilated cervix can leave

the relatively larger head trapped above the cervix. Tis is more

likely when disproportion between the head and body is signi-

cant. Examples are preterm or growth-restricted etuses or those

with macrocephaly rom hydrocephaly. Second, umbilical cord

prolapse is an ever-present risk. Last, twin etuses may become

locked together during delivery i the rst presents breech and

the second cephalic. As the breech o the rst twin descends

through the birth canal, the chin locks between the neck and

chin o the second cephalic-presenting co-twin. Tis phenomenon is rare, and Cohen and coworkers (1965) described it only

once in 817 twin gestations.

I these problems are anticipated or identied, cesarean

delivery is oten preerred with a viable-sized etus. Even without these problems, many obstetricians perorm cesarean delivery i the rst twin presents as breech, and this is our practice.

However, data support the saety o vaginal delivery or twins

older than 32 weeks and weighing >1500 g (Blickstein, 2000).

In one study, cesarean delivery was planned in 1169 such pairs.

Vaginal delivery was planned in 298 pairs and was successul in

64 percent. Cesarean delivery o the second twin was done in

1 percent. Neonatal mortality or morbidity measures did not

dier between delivery groups (Korb, 2020).

■ Labor Augmentation or Induction

In general, active labor with twins progresses more slowly in

both nulliparas and multiparas compared with that in singletons (Hochler, 2021). Second-stage labor o the rst twin also

is longer (Levin, 2021).

In women with twins who meet all criteria or oxytocin

administration, labor augmentation is suitable (Chap. 26, p.

492). For labor induction, studies have ound that oxytocin

alone or in combination with cervical ripening can saely be

used in twin gestations (Hamou, 2016; Ko, 2014). Compared

with prelabor cesarean delivery, others have ound higher

maternal morbidity rates with labor induction, and ultimate

cesarean delivery rates approximate 40 percent (Dougan, 2020;

Grossman, 2021). In an analysis o twin births in the United

States, induction rates o twin pregnancies have declined rom

nearly 14 percent in 1999 to 10 percent in 2008 (Lee, 2011).

Generally, at Parkland Hospital we do not augment or induce

labor in women with a multietal gestation. Concerns include

risks or uterine rupture rom an overdistended uterus and postpartum hemorrhage. In suitable candidates with a strong desire

or vaginal birth, amniotomy induction has been one option.

■ Vaginal Delivery

Ater delivery o the rstborn, one clamp is placed near the

neonate, and another is placed nearer the placenta. Until the

last etus is delivered, each cord must remain clamped to prevent etal hypovolemia and anemia caused by blood leaving

the placenta via anastomoses and then through an unclamped

cord. Cord blood is generally not collected until ater delivery o all etuses. Ater the second neonate is delivered, two

plastic clamps are placed on the placenta’s cord to dierentiate it rom the rst. In higher-order deliveries, color-tagged

or alphabetically labeled clamps can be simpler than adding

additional clamps. Tis same practice holds or cesarean delivery. At this time, evidence is insufcient to recommend or or

against delayed umbilical cord clamping in multietal gestations

(American College o Obstetricians and Gynecologists, 2020a).

Following vaginal delivery o the rst twin, the presenting

part o the second twin, its size, and its relationship to the birth

canal should be quickly ascertained by combined abdominal,

vaginal, and, at times, intrauterine examination. Sonography is

a valuable aid. I the etal head or the breech is xed in the birth

canal, moderate undal pressure is applied and membranes are

ruptured. Immediately aterward, digital examination o the

cervix is repeated to exclude cord prolapse. Labor is allowed to

resume. I contractions do not begin within approximately 10

minutes, dilute oxytocin may be used to stimulate contractions.

Te preerred interval between delivery o the rst and second twins is requently cited as <30 minutes. In some studies,

longer intertwin intervals are associated with poorer outcome

o the second twin (Leung, 2002; Stein, 2008). Others have

correlated etal heart rate tracing abnormalities during the

intertwin interval rather than its length with poorer outcome

(Algeri, 2019).

I the occiput or breech presents immediately over the pelvic

inlet, but is not xed in the birth canal, the presenting part can

oten be guided into the pelvis by one hand in the vagina, while

a second hand on the uterine undus exerts moderate pressure

caudally. A presenting shoulder may be gently converted into a

cephalic presentation. Alternatively, with abdominal manipulation, an assistant can guide the presenting part into the pelvis.

Sonography can aid guidance and allow heart rate monitoring.

I the occiput or breech is not over the pelvic inlet and cannot be so positioned by gentle pressure or i appreciable uterine

bleeding develops, delivery o the second twin can be problematic. o obtain a avorable outcome, an obstetrician skilled in

intrauterine etal manipulation and anesthesia personnel skilled

in providing anesthesia to eectively relax the uterus or vaginal

delivery o a noncephalic second twin are essential (American

College o Obstetricians and Gynecologists, 2021b). o take

maximum advantage o the dilated cervix beore the uterus contracts and the cervix retracts, delay should be avoided. Prompt

cesarean delivery o the second etus is preerred i no one present is skilled in the perormance o internal podalic version or

i anesthesia that will provide eective uterine relaxation is not

immediately available.

■ Trial of Labor after Cesarean Delivery

Te main concern with trial o labor ater cesarean delivery

(OLAC) is uterine rupture rom a distended uterus. One

metaanalysis ound rates o rupture and o successul vaginal

birth were comparable with those or OLAC in singleton gestations (Kabiri, 2019). Other studies also support the saety o

OLAC or selected women with twins (Cahill, 2005; Ford,

2006). In one assessment o 186 women undergoing OLAC,

two thirds delivered both twins vaginally. O ailed attempts,

45 percent underwent cesarean or delivery o the second twin

(Varner, 2005). According to the American College o Obstetricians and Gynecologists (2019), evidence currently does not

suggest an increased risk o uterine rupture, and women with

twins and one previous cesarean delivery with a low transverse

incision may be considered OLAC candidates. At Parkland

Hospital, we recommend repeat cesarean delivery.

■ Cesarean Delivery Technique

Several unusual intraoperative problems can arise during cesarean delivery o twins or higher-order multiples. Supine hypotension is common, and thus gravidas are positioned in a let

lateral tilt to deect uterine weight o the aorta. A low transverse hysterotomy is preerable i the incision can be made large

enough to allow atraumatic delivery o all etuses. Piper orceps

can be used i a second twin is presenting breech. In some cases,

a vertical hysterotomy beginning as low as possible in the lower

uterine segment may be advantageous. For example, i a etus

is transverse with its back down and the arms are inadvertently

delivered rst, it is much easier and saer to extend a vertical

uterine incision upward than to extend a transverse incision

laterally or to make a “” incision vertically.

■ Triplet or Higherorder Gestation

Fetal heart rate monitoring during labor with triplet pregnancies is challenging. A scalp electrode can be attached to the

presenting etus, but it is difcult to ensure that the other two

etuses are each being monitored separately. With vaginal delivery, the rst neonate is usually born with little or no manipulation. Subsequent etuses, however, are delivered according to

the presenting part. Tis oten requires complicated obstetrical maneuvers such as total breech extraction with or without

internal podalic version or even cesarean delivery. Associated

with malposition o etuses is an increased incidence o cord

prolapse. Moreover, reduced placental perusion and hemorrhage rom separating placentas are more likely during delivery.

Data rom the Consortium on Sae Labor ound that only 17

percent o women with triplet gestation attempting vaginal

birth were actually delivered vaginally. In these births, composite neonatal morbidity rates were increased (Lappen, 2016).

In 7000 triplet pregnancies, vaginal delivery also was associated with a higher perinatal mortality rate (Vintzeleos, 2005).

For all these reasons, we deliver most pregnancies complicated

by three or more etuses by cesarean delivery. Vaginal delivery is reserved or those circumstances in which survival is not

expected because etuses are markedly immature or abnormal

or maternal complications make cesarean delivery hazardous

to the mother. Centers or Disease Control and Prevention

(2009) national data show that 94 percent o triplets are delivered by cesarean.

Other cases series describe more positive outcomes (Grobman, 1998; Peress, 2019). In sum, the American College o

Obstetricians and Gynecologists (2021b) notes vaginal birth

may be considered or uncomplicated triplet pregnancies, i

the rst etus is cephalic and attendants are experienced with

multietal vaginal birth. As with twins, candidate etuses should

weigh >1500 g.

REDUCING FETAL NUMBER

For multietal gestations, multietal pregnancy reduction (MPR)

aims to lower etal number to improve survival rates o the

remaining etuses. Instead, with selective reduction, early pregnancy intervention ocuses on a etus with an anomaly or serious health risk. With selective termination, the indications are

analogous to those or selective reduction, but interventions

are perormed at a later gestational age (American College o

Obstetricians and Gynecologists, 2020c).

Selective reduction or MPR is typically done in the late rst

trimester. Tis gestational age is chosen because most spontaneous abortions have already occurred, the remaining etuses are

large enough to be evaluated sonographically, the amount o

devitalized etal tissue remaining ater the procedure is small,

and the risk o aborting the entire pregnancy as a result o the

procedure is low.

Skill in sonographically guiding needles through the mother’s abdomen and uterus is required. For reductions o etuses

with their own chorion, potassium chloride is used and is

injected into the etal heart or thorax. Entering or traversing the

sacs o etuses picked or retention is avoided. For monochorionic etuses and potentially shared vasculature, cord-occlusion

is used, and with radiorequency ablation (RFA), the needle is

inserted into the intraetal portion o the umbilical cord.

Beore reduction procedures, discussion should include the

morbidity and mortality rates associated with expectant care,

entire pregnancy termination, or selective etal reduction (American College o Obstetricians and Gynecologists, 2020c). With

these options, specic risks include (1) loss o all remaining

etuses, (2) abortion or retention o the wrong etus, (3) damage

without death to a etus, (4) preterm labor, (5) etuses with discordant or growth restriction, and (6) maternal complications.

With reduction procedures, uncommon potential complications are inection, hemorrhage, or disseminated intravascular

coagulopathy because o retained products o conception.

■ Multifetal Pregnancy Reduction

In most cases o MPR, higher-order gestations are reduced

by one or more etuses. With triplets, reduction to twins or

a singleton lowers the rate o preterm birth beore 34 weeks

compared with expectant management. Miscarriage rates are

not higher (Anthoulakis, 2017; Morlando, 2015). With even

higher-order multiples, spontaneous loss and preterm birth

rates also decline ater MPR (Evans, 2014; Liu, 2020).

win gestations also may be reduced to a singleton pregnancy. Maternal comorbidities or concerns or monochorionic

twin complications, described earlier, are requent indications

(Rao, 2021; Vieira, 2019).

■ Selective Reduction or Selective Termination

I multiple etuses are discordant or anatomical or genetic

anomalies, elimination o the abnormal etus is an option.

Other indications are severe S, APS, RAP, or sFGR.

Because abnormalities are oten not ully delineated until the

second trimester, selective termination is perormed later in

gestation than selective reduction and entails greater risk. Tis

procedure is thereore usually not perormed unless the abnormality is severe but not lethal. In some cases, termination is

considered because the abnormal etus may jeopardize the normal one

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