CASE 80: LABOUR
History
A 22-year-old woman in her second pregnancy has arrived on the labour ward at 38 weeks
3 days. She had a normal delivery 18 months ago. This pregnancy has been complicated by
persistent vomiting until 20 weeks and more recently by anaemia. She reports contractions
commencing approximately 4 h ago. She took paracetamol at home and tried to relieve the
pain with a bath, but now feels she cannot cope with the pain.
She had a show 2 days ago but has had no bleeding since then and has not noticed any vaginal
leak. She has felt the baby moving normally all day.
Examination
The blood pressure is 110/58 mmHg and heart rate is 98/min. The presentation is cephalic
with 2/5 palpable abdominally. Uterine contractions are palpable and the uterus is nonirritable. On vaginal examination the cervix is 5 cm dilated and the head is 1 cm above the
ischial spines. The fetal position is right occipitotransverse with mild caput and moulding. The membranes are intact but rupture spontaneously during examination, with clear
liquor draining.
The woman requests an epidural for pain relief and is therefore commenced on continuous
cardiotocograph monitoring. After 20 min you are called in to review the situation.
INVESTIGATIONS
The CTG as you walk in is shown in Fig. 80.1.
Questions
• Describe the CTG.
• What are the possible causes of this CTG?
• What management would be appropriate now?
20:20
200
180
160
140
120
100
80
60
HR
200
180
160
140
120
100
80
60
HR
20:10 20:30
200
180
160
140
120
100
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100 HR
75
50
25
UA
100
75
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25
UA
Figure 80.1 Cardiotocograph.100 Cases in Obstetrics and Gynaecology
224
ANSWER 80
CTG interpretation
The initial 15 min of CTG shows a baseline of 145/min with normal variability (12/min) and
no visible acceleration or decelerations. Following this there is a drop in fetal heart rate to
70/min for 7 min before gradual recovery to 125/min. Contractions are 2 in 10 until the tocograph becomes unreadable.
This is a previously low-risk pregnancy and this CTG shows a fetal bradycardia (reduction in
baseline heart rate to below 100/min). In many cases no cause is identified.
! Causes of fetal bradycardia
• Placental abruption
• Uterine rupture
• Maternal hypotension (e.g. after epidural insertion)
• Bleeding vasa praevia
Management
The ‘rule of 3s’ should be employed in managing this woman:
• If the deceleration has not recovered at 3 min call for help.
• If the deceleration has not recovered at 6 min transfer to theatre and prepare for
immediate delivery.
• If the deceleration has not recovered at 9 min deliver immediately by category one
(‘crash’) caesarean section (if immediate instrumental vaginal delivery is not possible). This will usually involve general anaesthetic unless an effective spinal anaesthetic is achievable by an experienced anaesthetist in a similar time.
The labour ward theatre team should be called (including anaesthetist, obstetric registrar,
paediatrician, midwife in charge, theatre staff) and the woman transferred to the operating theatre. On occasion the bradycardia recovers as preparation is underway for the caesarean, in which case the plan may be reviewed. Otherwise the baby should be delivered
immediately.
In this case the bradycardia did not recover and the baby was delivered within 12 min of the
decision being made. No cause was found for the bradycardia at caesarean section.
INVESTIGATIONS
The umbilical artery cord blood analysis at delivery was:
Artery Vein
pH 7.06 7.23
pCO2 8.20 mmHg 6.30 mmHg
Base excess –6.4 mmol/L –5.2 mmol/LCase 80: Labour
225
The baby initially made poor respiratory effort and had a heart rate less than 100/min, but
recovered quickly with drying and warming. The Apgar score for the baby was 5 at 1 min and
9 at 5 min.
KEY POINTS
• A prolonged bradycardia (>6 min) is an indication to transfer a woman to theatre
for consideration of immediate delivery if the heart rate has not then recovered
by 9 min.
• There is no place for a fetal blood sample in the management of fetal bradycardia.
• A cause is not always found for an abnormal CTG.
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