CASE 95: LABOUR
History
A 22-year-old woman is admitted to the labour ward for induction of labour at 40 weeks’
gestation. This is her first ongoing pregnancy, having had a first-trimester miscarriage 13
months previously. She booked at 9 weeks and had normal booking blood tests. The 11–14-
week scan and 21-week anomaly scan did not show any obvious fetal abnormality. Blood
pressure and urinalysis have always been normal.
At her 32-week midwife appointment she reported feeling very uncomfortable abdominally,
and the midwife measured the symphysiofundal height to be 36 cm. A further ultrasound
scan was therefore requested which showed normal fetal growth but increased liquor volume.
She had been reviewed in the antenatal clinic and was tested for gestational diabetes with
glucose tolerance test but this was normal. Subsequent examinations had again confirmed
an increased symphysiofundal height, and further ultrasound scan at 36 weeks again showed
normal growth, no fetal abnormality and markedly increased liquor volume. The fetal movements had always been normal.
A decision had been made for induction of labour at 40 weeks because the woman had become
so uncomfortable and breathless.
On palpation the fetus was cephalic with the head 4/5 palpable abdominally. Cardiotocograph
(CTG) was reassuring; 2 mg of prostaglandin gel had been inserted into the posterior fornix
of the vagina and CTG monitoring continued for a further 20 min.
The woman then mobilized and contractions started within an hour. She requested an epidural for analgesia and while this was being prepared CTG monitoring was commenced. At this
stage, spontaneous rupture of membranes occurred with a very large volume of clear liquor
soaking the bed sheets.
INVESTIGATIONS
The CTG is shown in Fig. 95.1.
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Figure 95.1 Cardiotocograph.100 Cases in Obstetrics and Gynaecology
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Questions
• Describe the CTG.
• What is the likely diagnosis?
• How would you confirm the diagnosis and manage this situation?Case 95: Labour
271
ANSWER 95
The CTG shows deep atypical variable decelerations. The sudden CTG deterioration after
rupture of membranes in a woman with polyhydramnios suggests the likelihood of prolapse
of the cord. Other possible diagnoses are placental abruption or fetal head compression from
precipitate labour. The diagnosis is easily confirmed with vaginal examination. A loop of
umbilical cord will be palpated in the vagina and will be pulsatile.
! Risk factors for cord prolapse
• Polyhydramnios
• Preterm delivery
• Malpresentation
• Unstable presentation
• Multiple pregnancy
This is an obstetric emergency and the emergency bell should be activated with the senior midwife, theatre team, senior obstetrician, paediatrician and anaesthetist summoned immediately.
The important management steps are:
• the examiner should not remove their fingers from the vagina and should attempt to elevate the fetal head above the cord and minimize contact with the cord to prevent spasm
• the woman should be rotated into the ‘all-fours position’ (head lower than buttocks),
which will facilitate relieving the weight of the baby and abdomen from the prolapsed cord
• she should be transferred to theatre immediately for caesarean section
• intravenous access should be obtained, and a general anaesthetic administered,
using a rapid sequence induction with cricoid pressure
• the examiner should only remove their fingers from the presenting part in the
vagina when the uterus has been opened and the baby is being delivered.
KEY POINTS
• Cord prolapse should be suspected in cases of fetal heart abnormality occurring
after rupture of membranes.
• It is an obstetric emergency and necessitates immediate caesarean section.
• Attempts should be made to minimize pressure on the cord pending delivery.
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